Cerebral hypoxia
- Article
- 2021-03-02
Cerebral hypoxia refers to a condition in which the oxygen supply to the brain decreases even though there is adequate blood flow. Drowning, strangulation, suffocation, suffocation, cardiac arrest, head trauma, carbon monoxide poisoning, and complications from general anesthesia can create conditions that can lead to cerebral hypoxia. Symptoms of mild cerebral hypoxia include inattention, poor judgment, memory loss, and impaired motor coordination. Brain cells are extremely sensitive to oxygen deprivation and can begin to die within five minutes after the oxygen supply is interrupted. When hypoxia persists for a long time, it can cause coma, seizures, and even brain death. In brain death, there is no measurable activity in the brain, although cardiovascular function is preserved. Life support is required for breathing.
Therapy
Treatment depends on the underlying cause of the hypoxia, but basic life support systems must be put in place: mechanical ventilation to secure the airways; fluids, blood products or medicines to support blood pressure and heart rate; and drugs to suppress seizures.
Prognosis
Recovery depends on how long the brain has been deprived of oxygen and how much brain damage has occurred, although carbon monoxide poisoning can cause brain damage days to weeks after the event. Most people who make a full recovery have only been unconscious for a short time. The longer someone is unconscious, the greater the risk of death or brain death and the smaller the chance of meaningful recovery. During recovery, psychological and neurological abnormalities such as amnesia, personality regression, hallucinations, amnesia and muscle spasms and twitches can occur, persist and then disappear.
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